Two specific intestinal bacteria that were identified as sects for multiple sclerosis – naturalnews.com

A pioneering new study revealed a direct link Between the specified intestine bacteria and the development of multiple sclerosis (MS)The increasing understanding that autoimmune diseases may arise in the intestine – not only in genetics or the environment alone. Posted in pnasThis research analyzed 81 pairs of identical twins, as only one brother had multiple sclerosis and found two bacteria, Eisenberga fits and Lachnoclostridium, It is constantly associated with the disease.

Main points:

• The researchers cultivated the intestinal microbes of mice twins, which then developed symptoms similar to multiple sclerosis.
• These bacteria flourish in the small intestine – a hot point for immunosuppressive – and may deceive the immune system to attack neurons.
• Women showed a higher allergy, reflecting the non -proportional MS effect on females.
• The results indicate that food interventions, such as promoting fiber intake, can starve harmful bacteria with beneficial microbes support.

Brain bowel connection: How can bacteria pay autoimmune

For years, scientists suspected that multiple sclerosis – the exhausted autoimmune condition where the immune system erodes the neurotoxicleum sheaths around the nerves – strong relationships with bowel health. But this study goes beyond the scientific relationship, which indicates that the specific bacteria can raise the disease directly. Researchers used stool implants from twins with multiple sclerosis in mice -free mice genetically vulnerable to nervous inflammation. Within weeks, these mice developed paralysis, while those who receive bacteria from healthy twins remained free of symptoms.

The study digs further, the study has defined the small intestine – an area full of immune cells – such as the battlefield that these immune bacteria manipulate. Eisenberga fits and Lachnoclostridium Feremal fibers and produce short sequential fatty acids (Scfas), which must be anti -inflammatory. But in multiple sclerosis, something goes there: their metaphysical activity may lead to an immune dysfunction instead.

Eisenberga offers:

Bacteria is less than FIRMICOTES Physlum, this strain is associated with MS in twin studies. It flourishes in the small intestine, where Dietary fiber fermentation in short sequential fatty acids (For example, acetate, propionate). While Scfas usually enhances the organizational TT (TREGS) to reduce inflammation, in MS, the E. Tayi receptors may enhance the TH17 cells instead-the inflammatory factors involved in autoimmune. Research indicates that E. Tayi may lead to the deterioration of Mucin, which weakens the intestine of the bowel barrier and allowing bacterial lipid sugars (LPS) to leak into blood circulation, and settling of physical inflammation.

Lachnoclostridium:

Gender Lachnospiaceae family, Popular to the production of butter. Ironically, some breeds (for example, L. Bolteae) are associated with MS, perhaps due to the effects of the dynasty or reactions based on the context. In multiple sclerosis Lachnoclostridium It may be transformed from the production of anti -inflammatory butter to the infections supportive under low fiber conditions. Some species resign tribetovan in the derivatives of the endolis, which can cross the blood barrier in the brain and adjust nervous inflammation through the signs of hydrocarbon receptors (AHR)-an unorganized path in MS.

Gevosos, antibiotics and modern diets disrupt our bowel balance

While genetics and viruses such as Epstein Bar can prepare a person to multiple nerve sclerosis, the explosion of autoimmune diseases in industrialized countries indicates that environmental toxins and treated diets play a major role. GvosateIt turns out that the active ingredient in Roundup, disturbing the bacteria of the intestine, especially those that digest the fibers – from a government point of view, the same useful strains (Lachnospiraceae) that were associated with MS in this study.

The modern Western diet – in the fibers, may suffer from emulsions, which are loaded with pesticides – may be hungry with good bacteria while feeding it harmful. Can it help restore balance through fiber -rich foods (legumes, whole grains and peaches) in suppressing the ms microbes?

This leading study is not only enhanced the role of the intestine in multiple nerve sclerosis, but also the doors of the target treatments for microbium. The causal link – which was distinguished by stool transplants in mice – struggles that autoimmune players are adjustable, providing hope for environmental interventions such as precision probiotics or treatment for eliminating harmful strains. Meanwhile, food strategies (for example, high -icenia, rich in fillen) may starve bacteria that support inflammation during care Lactobacillus and bifidobacteriumWhich is a TH17 meter dominance.

It is important, the study highlights the sensitivity of sex, as female mice reflect the spread of MS from 3: 1 to males in humans-and perhaps it is associated with adjusting estrogen from the permeability of the intestine. Moreover, it challenges the hypothesis of hygiene, which suggests that industrial lifestyles (for example, glephos, ultra -treated foods) disrupt microbial harmony, create a lenient environment for Autoimmune.

The recovery of Dr. Wales-which was directed via a Balo-inspired protocol is rich in vegetables that contain sulfur and organs-is considered the possibilities of nutritional programming to rebuild flexible microbium. Future research should explore bacterial federations instead of individual guilty, as the environmental balance – not the judiciary – carries the key. While we verify the legacy of the immune intestine, one fact crystallizes: the MS administration may be wet close to immunodeficiency to microbial reconciliation.

Sources include:

Exposing news.com

pnas.org

Enok, Brighton